Disrupted fetal carbohydrate metabolism in children with autism spectrum disorder

Table 1 Cohort characteristics

	Neurotypical (n = 36)	ASD (n = 16)	P-value
BMI at New OB visit (kg/m²)	33.43 ± 10.21	30.38 ± 6.05	0.38
Gestational age (weeks)	38.33 ± 1.43	38.51 ± 1.81	0.73
Birth Weight (g)	3275.11 ± 497.05	3501 ± 816.41	0.29
Ethnicity (% Hispanic or Latino)	3%	14%	0.14
Race (% non-white)	3%	21%	0.06
Infant Sex (% male)	42%	77%	0.03
Maternal age (years)	28.18 ± 8.15	24.89 ± 10.62	0.23
APGAR at 1 min (median)	8.00 ± 1.67	8.78 ± 0.44	0.18
APGAR at 5 min (median)	8.67 ± 0.76	8.93 ± 0.27	0.22
Cesarean Delivery (%)	25%	29%	0.80
Gravida	3.03 ± 1.93	3.3 ± 1.95	0.70
Parity	1.42 ± 1.23	1.40 ± 1.71	0.96

Cord blood samples were analyzed from pregnancies resulting in a child without any diagnosed neurodevelopmental disorder (neurotypical) or with Autism Spectrum Disorder (ASD). Only male sex was significantly different between groups (more prevalent in ASD). Values reflect mean ± standard deviation unless otherwise noted. Categorical variables were compared via chi-square. Continuous variables were compared via Student’s t-test. P < 0.05 was significant. Bolded findings are statistically significant

ISSN: 1866-1955